Abstract (Source: European Eating Disorders Review)MedWorm Message: Register for MedMatcha, MedWorm's medical advertising network, and receive $5 free advertising.

 What does it mean to create a character for oneself, a character that isn't just defined by an eating disorder? Does anyone really construct a character, or is this something one rather falls into, or lets emerge?These are questions I've been musing on over the past fortnight. My boyfriend and I have just returned from ten days travelling through Italy and Switzerland with my father and his girlfriend, in his camper van - an impressive vehicle with two double beds inside and recently added black cow spots outside. We spent the first day lounging by a backwater of Lake Como, with sun pouring down and mountains all around - the other campers mostly foreigners, Russians, Germans, and Dutch, but a few Italian men and girls wandering around, with an insouciance that belonged to a differen...

How to express your concernSource: HealthDay Related MedlinePlus Page: Eating Disorders (Source: MedlinePlus Health News)

How to express your concernSource: HealthDay Related MedlinePlus Page: Eating Disorders (Source: MedlinePlus Health News)

How to express your concernSource: HealthDay Related MedlinePlus Page: Eating Disorders (Source: MedlinePlus Health News)

How to express your concernSource: HealthDay Related MedlinePlus Page: Eating Disorders (Source: MedlinePlus Health News)

How to express your concernSource: HealthDay Related MedlinePlus Page: Eating Disorders (Source: MedlinePlus Health News)

Conclusions  Peripheral QCT traits are associated with common risk factors of osteoporosis in young Finns and discriminate between those with and without low-energy fractures. In men, trabecular bone loss at peripheral bone sites starts before the age of 40 years. Content Type Journal ArticleDOI 10.1007/s11657-010-0043-7Authors Marika M. L. Laaksonen, Department of Food and Environmental Sciences, University of Helsinki, Helsinki, FinlandHarri Sievänen, UKK Institute, Tampere, FinlandSanna Tolonen, Department of Food and Environmental Sciences, University of Helsinki, Helsinki, FinlandVera Mikkilä, Department of Food and Environmental Sciences, University of Helsinki, Helsinki, FinlandLeena Räsänen, Department of Food and Environmental Sciences, University of...

Last month I posted an essay linking the dramatic increase in diagnosed ADHD (Attention Deficit Hyperactivity Disorder) to our increasingly restrictive system of schooling (see ADHD and School). I presented evidence there that (a) the official, DSM-IV diagnostic criteria for ADHD focus primarily on school-related issues such as sitting in seat, completing assignments, and not interrupting teachers; (b) most diagnoses of ADHD begin with referrals from teachers or other school personnel; (c) teachers' ratings, if used alone, would produce far more ADHD diagnoses than is the case when those ratings are balanced by parents' ratings; and (d) the rapid increase in ADHD diagnoses occurred over the same period that high-stakes standardized testing increasingly dominated the school environment. My ...

Watching a child suffer with an eating disorder can be devastating for parents, especially if they're told they're to blame. The truth is, while no one fully understands what causes these illnesses, we do know that for children and teens with eating disorders, parents are usually a crucial part of the solution.Step one is recognizing the problem. Here are five warning signs that your child may be dealing with anorexia or another eating disorder.1. Weight stagnation or loss. Weight gain typically continues into the early 20s. So if your child or teen continues to grow but doesn't gain weight-especially in early adolescence, when most growth takes place-s/he may be in the early stages of an eating disorder. And any weight loss, even unintentional, may trigger an eating disorder in vulnerable...MedWorm Message: Register for MedMatcha, MedWorm's medical advertising network, and receive $5 free advertising.

Abstract Background and aims  Cancer cachexia, a complex wasting syndrome, is common in palliative medicine. Animal models expand our understanding of its mechanisms. A review of cancer cachexia and anorexia animal models will help investigators make an informed choice of the study model. Results and discussion  Cancer-anorexia cachexia animal models are numerous. No one is ideal. The choice should depend on the research question. To investigate cancer–anorexia cachexia independent of pro-inflammatory cytokine effects, the MAC16 ADK and XK1 are useful. MAC16 ADK helps study the host’s tumor metabolic effects, independent of any anorexia or inflammation. XK1 is both anorectic and cachectic, but data about it is limited. All other models induce a host i...

Authors: Tachibana T, Matsuda K, Sawa H, Mikami A, Ueda H, Cline MA Neuromedin B (NMB) and neuromedin C (NMC) are homologues of bombesin and are distributed throughout both the brain and gastrointestinal tract. The physiological roles of these bombesin-like peptides in chicks (Gallus gallus) have not been documented. Therefore, the purpose of the present study was to measure the effects of these bombesin-like peptides on food intake, crop-emptying rate and body temperature in chicks, and then to compare these effects with those of bombesin. Intracerebroventricular (ICV, 5 nmol) and intraperitoneal (IP, 300 nmol/ kg) injections of NMB, NMC, and bombesin significantly decreased food deprivation-induced food intake. When ICV injected (5 nmol), all 3 peptides significantly reduced crop-emp...

We have shown reduced failure load and stiffness of the distal radius in women with anorexia nervosa (AN) compared with age-matched y control subjects by using finite element models created from 150-µm resolution flat-panel volume CT; reductions in trabecular bone volume fraction, trabecular number, and bone mineral density and an increase in trabecular separation were observed in the AN group. (Source: Continuous Publishing articles)

In September, 2009, a 31-year-old woman presented to our emergency room with a 3-day history of severe abdominal pain. 3 weeks before admission she had undergone a caesarean section to deliver her second child. She had no nausea, anorexia, vomiting, or fever. Examination was normal apart from a positive Murphy's sign. C reactive protein (CRP) concentrations were raised at 9·7 mg/L, but numbers of neutrophils were normal. We suspected acute cholecystitis and started antibiotic treatment with amoxicillin; however, ultrasonography showed no signs of an inflamed gallbladder, and a CT scan of the abdomen showed signs only of peritonitis and postoperative changes. (Source: LANCET)

This report discusses a clinically relevant pharmacokinetic CYP3A4 drug-drug interaction between sorafenib and felodipine in an 80-year-old Caucasian patient with HCC. On day 15, after the introduction of sorafenib (400 mg bid), sorafenib plasma concentration was at 3.6 mg/L. Felodipine (5 mg bid), an anti-hypertensive agent that is exclusively CYP3A4 substrate, was then introduced due to grade 2 sorafenib-related hypertension. On day 30, hypertension was well controlled. However, sorafenib plasma concentration was 3-fold greater (11.4 mg/L) and the patient experienced grade-3 anorexia. Since neither diarrhea nor cutaneous side effects were noticed at this time, sorafenib treatment was continued at the same daily dosage. On day 45, sorafenib plasma concentration w...MedWorm Message: Register for MedMatcha, MedWorm's medical advertising network, and receive $5 free advertising.

The Not-So-Obvious Signs You Might Miss (Source: Health News: CBSNews.com)

The Not-So-Obvious Signs You Might Miss (Source: Health News: CBSNews.com)

The Not-So-Obvious Signs You Might Miss (Source: Health News: CBSNews.com)

The Not-So-Obvious Signs You Might Miss (Source: Health News: CBSNews.com)

The Not-So-Obvious Signs You Might Miss (Source: Health News: CBSNews.com)MedWorm Message: Register for MedMatcha, MedWorm's medical advertising network, and receive $5 free advertising.